Collectively these final results recommend that while IFN-b creation was inhibited by H3N2, the manufacturing and activity of constitutive IFN-b collectively with viral recognition was enough to set off host cell apoptosis and suppress viral replication

H3N2 replication and IFN-b release pursuing protein synthesis inhibition with cycloheximide in Calu-3 cells and pBECs. Cycloheximide was extra to Calu-three cells and pBECs to inhibit protein synthesis 30 min…